Pharmacology & Toxicology Graduate Students
Andrew King, Graduate Student
2001, DVM Equivalent, Veterinary Medicine, Murdoch U/Australia
2004-present, Doctoral Candidate, Pharmacology & Toxicology
[First-year Student – Currently in Research Rotations]
   Contact Info:kingand@msu.edu
Selected Achievements and Awards:

Pfizer Safety Sciences Training Fellowship – 2005 -
Society for Experimental Biology and Medicine Young Investigator Award – Experimental Biology 2006
5th Hypertension Summer School 2005 – Travel award

Selected Publications/Abstracts

King, A.J., Fink, G.D. Chronic low-dose angiotensin II infusion increases venomotor tone by neurogenic mechanisms. Hypertension, 2006;48(5):927-933.

King, A.J., Bailie, M and Olivier N.B.. The magnitude of error introduced by application of heart rate correction formulas to the canine QT interval. Annals of Noninvasive Electrocardiology, 2006;11(4):289-298.

King, A.J., Davies, D.R. and Irwin, P.J.. Feline multiple myeloma: Literature review and four case reports. Australian Veterinary Practitoner. 2002; 32(4):146-151.

“Blood Pressure Measurement Techniques for Cardiovascular Risk Assessment”. Safety Pharmacology Society Meeting 2006, San Diego.

“Blood Pressure Measurement Techniques in Experimental Animals”. Data Sciences International; Rodent Telemetry Animal Models; 1 day Instructional and Wet Lab Educational Course. Michigan State University. August 22, 2006.

Andrew King, Melissa Li and Gregory Fink. The splanchnic circulation as a critical target for chronic low dose angiotensin II hypertension. Council for High Blood Pressure Research, San Antonio, 2006. (Oral Presentation).

Andrew King, N.B. Olivier, P.S. MohanKumar, J.S. Lee, V. Padmanabhan and G.D. Fink. Hypertension caused by prenatal testosterone excess in female sheep. Council for High Blood Pressure Research, San Antonio, 2006.

Andrew King and Gregory Fink. Chronic-low dose angiotensin II (AngII) increases venomotor tone by neurogenic mechanisms. Experimental Biology, San Francisco, 2006.

Andrew King, Courtney Zorn and Gregory Fink. The hypertensive response to chronic low-dose Angiotensin II (AngII) is dependent on arterial pressure (AP) measurement method and salt intake. Experimental Biology, San Francisco, 2006.

C. Northcott, A. King, G. Fink and J.R. Haywood. Angiotensin II causes elevated superoxide levels in the paraventricular nucleus during hypertension. Council for High Blood Pressure Research, San Antonio, 2006. (Oral Presentation).

J. Osborn, G. Fink, A. Sved, G. Toney and M. Raizada, A. King and P. Guzman. Characterization of the angiotensin II (AngII)-induced model of hypertension in rat for inter-laboratory investigation. Experimental Biology, San Francisco, 2006.