Selected Achievements and Awards:
Michigan State University Center for Integrative Toxicology travel award for SOT annual meeting, 2005, 2007 and 2008
Drug Discovery Toxicology specialty section of Society of Toxicology best poster award, 1st place, 2007
ASPET Division of Toxicology best paper award, 2nd place, 2007
Michigan Society of Toxicology graduate student poster award, 1st place, 2007
Comparative and Veterinary Specialty Section of Society of Toxicology graduate student poster award, 1st place, 2008
ASPET Division of Toxicology best paper award, 3rd place, 2008
Selected Publications/Abstracts:
Prior to entering program
Periyannan, G., Shaw, P., Sigdel, T., and Crowder, M.W. (2004). In vivo folding of recombinant Metallo-ï¢ï€lactamase L1 requires the presence of Zn (II). Protein Science 13: 2236-2243.
Since entering program
Luyendyk, JP., Shaw, PJ., Green, CD., Maddox, JF., Ganey, PE., and Roth, RA. (2005). Coagulation-Mediated Hypoxia and Neutrophil-Dependent Hepatic Injury in Rats given Lipopolysaccharide and Ranitidine. JPET 314:1023-1031.
Shaw PJ, Luyendyk JP, Ganey PE, and Roth RA. Hypoxia potentiates hepatocellular killing by neutrophil mediators in vitro. Toxicol. Sci., The Toxicologist 90: 440; 2006.
Shaw PJ, Ganey PE, and Roth RA. Co-exposure of mice to trovafloxacin and lipopolysaccharide leads to an altered inflammatory response in a mouse model of idiosyncratic liver injury. The Toxicologist 96: 624; 2007.
Shaw PJ, Fullerton AM, Ganey PE, and Roth RA. Co-exposure to trovafloxacin and lipopolysaccharide causes tumor necrosis factor alpha-dependent liver injury: a murine model of idiosyncratic liver injury. The FASEB Journal 21, 6: 867.14; 2007.
Shaw PJ, Hopfensperger M, Ganey PE, and Roth RA (2007). Lipopolysaccharide and trovafloxacin coexposure in mice causes idiosyncrasy-like liver injury dependent on tumor necrosis factor-alpha. Submitted to Toxicological Sciences.